Into morphologically normal adults. Thus, Roc2-Cul5 is not required for development. It is possible that Roc2-Cul5 is redundant with other CDL. A recent paper showed that Roc1-Cul2 and Roc2-Cul5 complexes may act redundantly during meiotic cell cycle progression in C. elegans [39]. RNAi knockdown of Roc2 or Cul5 did not reveal an obvious phenotype, consistent with our results. However, RNAi knockd


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